W 4-01WHAT IS NEW WITH THE VULVAR TERMINOLOGY?

24. Vulvar diseases and neoplasia
J. Bornstein 1.
1Galilee Medical Center and Bar-Ilan University Faculty of Medicine (Israel)

Background / Objectives

The approach to vulvar disease, which has been changed lately, has led to the introduction of the new terminologies for vulvar conditions: The IFCPC clinical and colposcopic terminology, The ISSVD terminology of Vulvar Squamous Intraepithelial Lesions (Table 1) and the consensus terminology of vulvar pain and vulvodynia (Tables 2 and 3).


Methods

Table 1:  2015 ISSVD Terminology of Vulvar Squamous Intraepithelial Lesions

Low grade squamous intraepithelial lesion of the vulva [Vulvar LSIL]
High grade squamous intraepithelial lesion of the vulva [Vulvar HSIL]
Vulvar Intraepithelial neoplasia [VIN], differentiated-type [DVIN]

 


Results

Table 2: 2015 Consensus terminology and classification of persistent vulvar pain and vulvodynia

 A. Vulvar pain caused by a specific disorder*

·                Infectious
·                Inflammatory
·                Neoplastic
·                Neurologic
·                Trauma
·                Iatrogenic
·                Hormonal deficiencies

B. Vulvodynia – Vulvar pain of at least 3 months' duration, without clear identifiable cause, which may have potential associated factors

              Descriptors:

·       Localized (e.g. vestibulodynia, clitorodynia) or Generalized or Mixed (localized and generalized)
·       Provoked (e.g. insertional, contact) or Spontaneous or Mixed (provoked and spontaneous)
·       Onset (primary or secondary) 
·       Temporal pattern (intermittent, persistent, constant, immediate, delayed)
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*Women may have both a specific disorder (e.g. lichen sclerosus) and vulvodynia


Conclusion

Table 3: 2015 Consensus terminology and classification of persistent vulvar pain and vulvodynia
Appendix: Potential factors associated with Vulvodynia*

·                Co-morbidities and other pain syndromes (e.g. painful bladder syndrome, fibromyalgia, irritable bowel syndrome, temporomandibular disorder) [LOE 2]
·                Genetics [LOE 2]
·                Hormonal factors (e.g. pharmacologically induced) [LOE 2]
·                Inflammation [LOE 2]
·                Musculoskeletal (e.g. pelvic muscle overactivity, myofascial, biomechanical)  [LOE 2]
·                Neurologic mechanisms:
Central (spine, brain) [LOE 2]
Peripheral – Neuroproliferation [LOE 2]
·                Psychosocial factors (e.g. mood, interpersonal, coping, role, sexual function) [LOE 2]
·                Structural defects (e.g. perineal descent) [LOE 3]
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*The factors are ranked by alphabetical order
LOE - Level of evidence


References